Kerr J



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Kerr J :



In een artikel in het gerenommeerde tijdschrift New Scientist maakten Britse onderzoekers afgelopen zaterdag melding van de vondst van genetische verschillen in witte bloedcellen tussen ME-patiŽnten en gezonde mensen [1]. Hiermee menen zij het bewijs te hebben geleverd van de biologische oorsprong van ME

Vermoeidheidssyndroom mogelijk toch ziekte  Bij patiŽnten met het chronisch vermoeidheidssyndroom (CVS) zijn duidelijke lichamelijke verschillen gevonden vergeleken met gezonde mensen.

Microbial infections in eight genomic subtypes of Chronic Fatigue Syndrome/Myalgic Encephalomyelitis (CFS/ME) 
Kerr, Main, Derek Enlander, David Honeybourne, Jon Ayres, David J Nutt and Jonathan Lihan Zhang, John Goudh, David Christmas, Derek Mattey, Selwyn Richards, Janice

Dr. Jonathan Kerr, principal investigator, St. George's Medical School"We’ve shown that several hundred genes are very, very different between patients and controls and we now must make sure that these are specific differences to this disease and after that we can use that information to design new treatments.”

ORIGINAL ARTICLE : Gene expression in peripheral blood mononuclear cells from patients with chronic fatigue syndrome

N Kaushik, D Fear, S C M Richards, C R McDermott, E F Nuwaysir, P Kellam, T J Harrison, R J Wilkinson, D A J Tyrrell, S T Holgate, J R Kerr

The Scottish Conservatives are pleased to make our time in the Parliament available to members to debate the Kerr report and the state of the national health service in Scotland. It is a matter of regret that the Scottish Executive refused to do so in its time prior to the recess, given the importance of the subject.

Gray matter volume reduction in the chronic fatigue syndrome


Cytokines in Parvovirus B19 Infection As an Aid to Understanding Chronic Fatigue Syndrome


concludes that patients with ME/ICD-CFS have reproducible alterations in gene regulation and that sixteen genes have an expression profile associated with the disorder that can be grouped according to immune, neuronal, mitochondrial and other functions.,,2-1702267,00.html

The analysis of 9,522 genes identified 35 that appeared to show differences, and a more precise examination of these revealed significant and genuine changes in the way that 16 of the genes were expressed in the CFS group.

Differences in gene expression have been found in the immune cells of people with the disease, a discovery that could lead to a blood test for the disorder

When a person is attacked by an infection some genes become very active while others shut down.  The overactive genes produce chemicals, which cause the symptoms of the illness.  Scientists carrying out this study will be examining the overactive genes to identify the chemicals that they are producing.  These chemicals may then be targeted by existing or novel drugs

Kerr’s team found that a group of genes in the white blood cells of CFS sufferers were up to four times more active than those without the affliction while one was less active.“The involvement of such genes does seem to fit with the fact that these patients lack energy and suffer from fatigue,”

 Scientists believe they have pinpointed biological markers of chronic fatigue syndrome which could help develop a test and treatment for the condition.

In as yet unpublished work, bona fide research has indicated that in those with ME/ICD-CFS there are more gene abnormalities present than are found in cancer sufferers.

The Centers for Disease Control recently published the results of their study of CFS patients in work titled "Utility of the blood for gene expression profiling and biomarker discovery in chronic fatigue syndrome"




PIONEERING research by Glasgow scientists which could lead to a cure for ME is under threat from a lack of funding.


 ME: 'Invisible disease' is now easier to read